TUDCA 500 mg Capsules

Research-grade capsule formulation supplied in a sealed bottle. TUDCA (tauroursodeoxycholic acid) is a conjugated hydrophilic bile acid studied in controlled laboratory settings for its relationship to ER stress modulation, hepatocellular protection, mitochondrial stability, apoptotic signaling, and bile-acid-associated cellular homeostasis.

Research Use Only

This product is intended strictly for laboratory and scientific investigation. Not for human consumption, therapeutic use, or veterinary application.

Specifications

Form

Capsules

Content

500 mg TUDCA per capsule

Quantity

60 capsules

Packaging

Sealed bottle

Storage

Store at room temperature, protected from light, keep desiccated

Molecular Formula

C₂₆H₄₅NO₆S

Molecular Weight

~515.7 g·mol⁻¹

IUPAC Name

2-[[(3α,5β,7β)-3,7-dihydroxy-24-oxocholan-24-yl]amino]ethanesulfonic acid

CAS Number

14605-22-2

Research Overview

TUDCA is studied as a bile-acid-derived research compound with strong interest in ER stress modulation, mitochondrial stability, and hepatocellular support pathways. In experimental settings, it is investigated for its relationship to unfolded protein response signaling, membrane stability, apoptosis-related mechanisms, cholestatic models, and broader cell-survival pathways. No therapeutic or clinical claims are made or implied.

Primary Research Areas

ER Stress & UPR Signaling

Experimental investigation of proteostasis, misfolded-protein burden, and unfolded protein response pathways including stress-adaptation signaling.

Hepatocellular Protection

Preclinical research focused on liver-cell integrity, bile-acid homeostasis, cholestatic processes, and hepatobiliary stress models.

Mitochondrial Function & Apoptosis

Laboratory studies examining membrane potential stability, cytochrome c release, apoptotic signaling, and cellular resilience under stress conditions.

Neuroprotection & Cell Survival Models

Research involving neuronal survival, stress-response signaling, and broader cytoprotective laboratory pathways beyond liver-focused models.